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c a neuropraxia, where there is focal or segmental demyelination with preservation of the axon and recovery in 2-12 weeks c an axonotmesis, where the axon is divided but the epineurium remains intact and regrows at 1 mm per day from the site of injury c a neurotmesis,where the nerve is divided and no longer in continuity with no recovery. damage being more extensive in axonotmesis than in neura- praxia, spontaneous regeneration is still possible, although longer, taking up weeks to months after the injury. In axo- notmesis, as well as in neurotmesis, a sequence of patho- logical events known as wallerian degeneration occurs in the nerve segment distal to the injury. Neurotmesis Neurotmesis (in Greek tmesis signifies "to cut") is part of Seddon's classification scheme used to classify nerve damage. It is the most serious nerve injury in the scheme. In this type of injury, both the nerve and the nerve sheath are disrupted. While partial recovery may occur, complete recovery is impossible. Contents 1 Symptoms Axonotmesis • Axonal injury with subsequent degeneraon and regeneraon • Occurs without disrupLon of endo/peri/ epineurium • IniLal anesthesia with Tinel's sign • Complete recovery within 12 months, onset of sensory return within 2-4 months (Sunderland 2nddegree injury) Neurotmesis "neurapraxia," "axonotmesis," and "neurotmesis" to describe peripheral nerve injuries.35 Neurapraxia is a comparatively mild injury with motor and sensory loss but no evidence of Wallerian degeneration. The nerve distally conducts normally. Focal demyelin-ation and/or ischemia are thought to be the etiolo-gies of the conduction block. Axonotmesis the axon and myelin is disrupted here, but the neuronal connective tissue remains intact (endo-, peri-, epineurium) more severe crush injury than that which causes neuropraxia Wallerian degeneration occurs because of loss of axoplasmic flow Electrodiagnostic findings: (click the image to the right) Axonotmesis neurotmesis neuropraxia pdf In neuropraxia, an injury to your peripheral nerve(s) causes symptoms like burning, stinging and pain. These mild nerve injuries typically heal on their own with rest and time. There are two commonly used classification for PNI- the Seddon Classification and the Sunderland Classification. Seddon classified nerve injuries into three major groups: neurapraxia, axonotmesis, and neurotmesis whereas Sunderland expanded Seddon's Classification to five degrees of peripheral nerve injury as described in the table below: [2] [3] as Seddon's classification. These include neuropraxia, axonotmesis and neurotmesis [3]. The brief description of these injuries with their consequences is given in table 1. Table 1. Seddon's Classification - General Features Neuropraxia Axonotmesis Neurotmesis There is an occurrence of paralysis but the peripheral degradation is absent [11] . The lowest degree of nerve injury in which the nerve remains intact but signaling ability is damaged is called neurapraxia. The second degree in which the axon is damaged but the surrounding connecting tissue remains intact is called axon